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Pathology Chapter 2

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Section 1

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granulomatous inflammation

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Pathology

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6 years ago

Date created

Mar 1, 2020

Cards (19)

Section 1

(19 cards)

granulomatous inflammation

Front

form of distinct chromic inflammation considered immature connective tissue that swells certain infections can stimulate this response such as TB

Back

1. red 2. swelling 3. heat 4.pain 5. loss of function

Front

local signs of inflammation

Back

hyperplasia

Front

increase number of cells

Back

regeneration- just as healthy as previous tissue repair- less healthy as previous tissue (Scars)

Front

difference between regeneration and repair?

Back

atrophy

Front

decrease in size or function of a cell, tissue, or organ

Back

PMN

Front

-first to immigrate to injury site. first line of defense cells -from bone marrow - 60-70% of all WBC -phagocytize (kill) tissue debris and pathogen

Back

bruxism

Front

grinding or clenching

Back

bruises, cuts, getting a bee sting

Front

examples of acute inflammation?

Back

calculus, heavy deposits, ill-fitting dentures

Front

examples of chronic inflammation?

Back

hypertrophy

Front

increase in size of individual cells

Back

tertiary intention

Front

infection at injury site, may cause primary healing to become secondary healing. infected injury must be left open until infection is controlled.

Back

primary intention

Front

type of repair little loss of tissue, clean edges can be joined, little granulation tissue

Back

intact skin or mucosa, stomach acid, tears, saliva, urine, inflammation!!!!!!

Front

innate defenses include?

Back

6 weeks-any movement of bones will delay healing time

Front

how long does bone take to repair?

Back

attrition

Front

wearing of tooth structure during mastication DOES NOT INCLUDE BRUXISM

Back

1. fever 2. leukocytosis- increase of WBC 3. lymphadenopathy 4. granulomatous inflammation

Front

systemic signs of inflammation?

Back

secondary intention

Front

some loss of tissue, edges cannot be joined together, large clot produces large amount of granulation. excessive scaring, might produce a KELOID

Back

monocyte (macrophage)

Front

-second cell to emigrate to injury site -from bone marrow -3-8% of all WBC -phagocytosis in addition to some immune responses

Back

edema and/or infection

Front

what may delay healing of bone?

Back