steriod binds to things that are transcription factors inside the cell
Back
Gi
Front
inhibits adenylate cyclase
Back
M2 and M4
Front
relaxation or inhibition, decrease cAMP, decreaes Ca2+ and increase K+
Back
Gigantism
Front
too much GH in childhood
Back
Goiter
Front
iodine deficiency
Back
laminar flow
Front
stream lined
Back
ductus arteriosus
Front
ligamentum arteriosum
Back
atripone
Front
muscarinic antagonist
Back
Section 4
(50 cards)
autoregulation
Front
maintenance of constant BF in the face of changing pressure
Back
angina pectoris
Front
inadequate BF to the heart
myocardial ischemia
Back
myogenic mechanism
Front
vascular smooth muscle contracts when stretched
Back
MRI
Front
magnetic field aligns H+ in tissues
no radation
Back
QRS
Front
depolarization of ventricles, triggering main pumping contractions
Back
heat exhaustion
Front
occurs in elevated air temp causes vasodilation and sweating
Back
T
Front
ventricular repolarization
Back
evaporation
Front
losing heat through conversion of water to gas
Back
potassium
Front
vasodilation
Back
conduction
Front
losing heat through physical interaction
Back
Progress zone
Front
rapid cell division without differentiation
Secretes FGF-10
Back
Active hyperemia
Front
increase in BF in response to increase metabolic activity
Back
heat stroke
Front
body temperature increases to a point of tissue damage
Back
adenosine
Front
vasodilation
Back
Sonic Hedgehog (Shh)
Front
induces Hox d expression
Induced by retinoic acid
deteremines which Hox d genes are produced
induces FGF-4 expression
Back
PR wave
Front
delay of AV node to allow filling of ventricles
Back
Hox d
Front
gives rise to specific structures in the limb
Back
Apical ectodermal redige
Front
secretes FGF-8 and FGF-4
maintains progress zone
required for limb bud outgrowth
Back
Coronary insufficiency
Front
caused by coronary artery disease
Back
aspirin
Front
cyclooxygenase inhibitor
reducing body temperature
Back
hypoxia
Front
induces pulmonary vascoconstriction
Back
Reactive hyperemia
Front
increase in BF in response to a previous reduction in BF
Back
protons
Front
vasodilation
Back
FGF-4
Front
proximodistal
posterior AER
maintains proliferation and inhibits differentiation in PZ
induced by shh
Back
nitroglycerin
Front
dilates coronary arteries
Back
red flare
Front
arteriolar dilation
Back
En1
Front
ventral signal
inhibits Wnt-7a expression
Back
malignant hyperthermia
Front
increase in metabolic rate, oxygen consumption, and heat production in skeletal muscle
heat dissipating mechanisms cannot keep pace
Back
Carbon dioxide
Front
vasodilation
Back
P wave
Front
depolarization of atria in response to the SA node triggering
Back
Digital Radiology
Front
digital film used with x rays
Back
CT Scan
Front
special x ray that gives a 3D cross section
lots of radiation
Back
Wnt 7a
Front
dorsal signal
important in AER formation
Back
pyrogens
Front
causes fever
Back
metabolic mechanism
Front
oxygen delivery equals oxygen consumption
Back
red line
Front
capillary dilation
Back
r-fng
Front
inhibited by En1
Back
convection
Front
losing heat through movement of air or water over body
Back
Zone of Polarizing Activity
Front
encodes posterior positional information
induces Shh
Back
chronic hypoxic vasoconstrction
Front
elevated pulmonary pressure
Back
wheal
Front
local edema caused by vasodilation of arterioles and vasconstriction of veins
Back
Retonoic Acid
Front
stimulates Shh production
Back
FGF-10
Front
proximodistal
PZ
maintains AER structure and function
Back
radiation
Front
heat loss through IR rays
Back
FGF-8
Front
proximodistal
produced by AER
maintains proliferation and inhibits differentiation in PZ
maintained by FGF-10
Back
Lmx1
Front
dorsoventral
induced by Wnt7a
specifies dorsal structure
Back
ST
Front
beginning of ventricular repolarization
Back
shivering
Front
rhythmic contraction of skeletal muscle
Back
oxygen
Front
vasoconstriction
Back
Fluoroscopy
Front
real-time visualization
Back
Section 5
(37 cards)
V2 antidiuretic hormone receptor
Front
found in renal collecting ducts
water reabsoprtion
maintains fliud osmolarity
Back
Arrector Pili Muscles
Front
muscles that induce the secretion of sebum
responsible for goosebumps
Back
Brain Natriurectic Peptide
Front
a hormone that comfirms there is a heart failure problem
produced by over stretching and enlarged hearts
Back
ventricular shunt lesion
Front
commonest shunt lesion
left to right
results in a step up of saturations in the pulmonary artery as compared to the right atrium
increased pulmonary blood flow, feeding difficulties, failure to thrive, cardiomegaly, murmur, no cyanosis
Back
Sweat glands
Front
glands that found everywhere in the body
deep and coiled
Back
Sebaceous glands
Front
glands that secrete sebum
attached to hair follicles
all over the body expect for palms of hands and feet
responsible for acne
Back
Tetraology of Fallot
Front
pulmonary stenosis
ventricular spetal defect
overriding aorta
right ventricular hypertrophy
Back
right ventricle
Front
coarsely trabeculated septal surface
valve attachments to ventricular septum
Back
Kerley's B lines
Front
a sign of plumonary edema on an X-ray; suggestive for a diagnosis of congestive heart failure
Back
shunt leasion
Front
a place where red and blue blood mix
Back
Errecrine glands
Front
secrete water-y sweat
respond to heat
Back
left ventricle
Front
smooth wall
fine trabeculae
valve with no septal attachments
Back
nitrates
Front
a type of heart failure drug that decreases the EDP but does not increase CO
causes wedge pressure
Back
Bohr Effect
Front
as partial pressure of oxygen increases, the percent saturation also increases
Back
heart failure
Front
inability of the heart to pump blood to meet the body's basic metabolic demands, or does so only with an elevated filling pressure
Back
digoxin
Front
a drug that improves the symptoms of heart failure because it is a weak inotrope
does not cure heart failure
Back
Eisenmenger's Syndrome
Front
sever pulmonary vascular disease from a long standing unrestrictive shunt
clinical cyanosis, polycythemia, and clubbing
Back
Angiotensin II
Front
increases secretion of aldosterone
Back
atrial septal defect
Front
blood shunting to right
TV pumps at normal pressure
problems in the closing of the foramen ovale
Back
dilated heart
Front
caused from systolic heart failure
Back
Human Umbilical Vein Endothelial Cells (HUVEC)
Front
most commonly used human vascular endothelial cell in research
Back
bicuspid aortic valve defect
Front
fusion between two of the three aortic valve cusps
most common congenital heart defect
Back
hypoplastic left heart syndrome
Front
CHD where there is a very very small LV
Back
beta-blockers
Front
a type of drug that helps attack the exessive increase in neuronal stimulation to the heat
negative inotrope, but takes the stress off the heart
Back
Starling's Law of the Heart
Front
change in SV in response to changes in VR
Back
prostacyclin
Front
modifies newborn pulmonary vascular resistance
inhibits clotting and vasodilation
Back
Vagus nerve (CN X)
Front
innervates the aortic arch baroreceptors
Back
Pulmonary edema
Front
leads to fluid in the lungs due to a high blood pressure
Back
Apocrine glands
Front
glands that become active at puberty
large, deep, and produce thick secretion
makes a smell
respond to stress and sexual activity
Back
digitalis
Front
a positive intropic drug
Back
precapillary sphincters
Front
smooth muscle that precedes capillaries
Back
ventricular septal defect
Front
left-to-right shunt lesion
Back
Glossopharyngeal nerve (CN IX)
Front
innervates the carotid sinus
Back
V1 Antidiurectic hormone receptor
Front
found in vascular smooth tissue
vasoconstriction of arterioles and increases TPR
Back
Patent Ductus Arterious
Front
caused by PGE2 and PGI2
derived form the 6th brachial arch vessel
problems in the closing of the ductus arteriosus