Section 1
Preview this deck
Rehabilitation
Front
| 5 | 0 | ||
| 4 | 0 | ||
| 3 | 0 | ||
| 2 | 0 | ||
| 1 | 0 |
Active users
0
All-time users
0
Favorites
0
Last updated
6 years ago
Date created
Mar 1, 2020
Cards (60)
Section 1
(50 cards)
Rehabilitation
Maximizing function of diseased tissues
Histamine
vasodilation, increased permeability of blood vessels
anaplasia
Undifferentiated cells with variable nuclear and cell structures
Sequelae
outcomes of primary condition; follow up consequences
Hyperplasia
increase number of cells-increase in tissue mass
microscopic level
requires microscope
prodromal period
Early development of a disease Signs nonspecific or absent
Etiology
cause of disease
neoplasm
new growth (tumor)
complications
New secondary or additional problems
Metaplasia
Mature cell type is replaced by a different mature cell type
mortality
the number of deaths in a population
precipitating factors
condition that triggers an acute episode
latent state
no symptoms or clinical signs evident
remission
signs and symptoms subside
Epidemiology
causative factors and track disease occurance
Hypertophy
increase in cell size-increase in tissue mass
incidence
number of new cases in a population within a specified time period
Prevalence
% of new and existing cases in a population within a time period
purulent
pus--thick and yellow green, more WBC's, cell debris, microorganisms
liquefaction necrosis
Dead cells liquefy due to release of cell enzymes
Signs of inflammation
redness, heat, swelling, pain, loss of function
hypoxia
decreased oxygen in tissues
epidemic
increase number of cases of an infectious disease occur in a given area
ischemia
decreased oxygen in cells
Gross level
visible to naked eye
predisposing factors
Tendencies that promote development of a disease in an individual
atrophy
decrease in cell size-decrease in tissue mass
convalescence
period of recovery
infarction
area of dead tissue due to lack of oxygen
chronic disease
develops slowly and lasts a long time
Fat necrosis
Fatty tissue is broken down into fatty acids
serous
clear, watery plasma--primary fluid, some proteints, WBC's
prognosis
probability for recovery or for other outcomes
autopsy
Examination of the body and organs after death
biopsy
excision of small amounts of living tissue
Diagnosis
identification of a disease
syndrome
collection of signs and symptoms
pandemic
increased number of cases across continents
acute disease
develops rapidly, lasts a short time
gangrene
necrotic or dead tissue caused by bacterial infection
Dysplasia
cells vary in size and shape within a tissue
Morbidity
number of people with a disease within a group
subclinical state
Pathologic changes occur, no obvious manifestations
Coagulative necrosis
Cell proteins are altered or denatured
fever
pyrexia
therapy
measures to promote recovery; slow disease progression
caseous necrosis
coagulation necrosis in which a thick, yellowish, "cheesy" substance forms
Pathogenesis
development of disease
fibrinous
thick and sticky--high cell and fibrin content
Section 2
(10 cards)