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Necrotic Lesion

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Pathology

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Last updated

1 year ago

Date created

Mar 14, 2020

Cards (95)

Section 1

(50 cards)

Necrotic Lesion

Front

Back

Fatty change

Front

Back

Pneumoconiosis

Front

Inhalation and retention of dust (Assocated with pulmonary fibrosis (radiograph) and tumors

Back

Haemoglobin

Front

Haemoglobinuria (Split red blood cells)

Back

Hyaline Degeneration

Front

Back

Ischaemia

Front

Intestinal Torsion

Back

Bilirubin

Front

Back

Fibrinous Inflammation

Front

Back

Fibrinoid degeneration

Front

Back

Mucoid Degeneration

Front

Back

Serous inflammation

Front

Blister

Back

Renal secondary hyperparathyroidism

Front

Back

Fatty change in liver

Front

Back

thrombosis and embolism

Front

blood clots inside intact blood vessels

Back

Coagulative Necrosis

Front

Canine herpes virus can cause this

Back

Haematin

Front

Back

Primary amyloidosis (AL)

Front

-Due to deposition of proteins from Ig Light chains. Can occur as a plasma cell disorder or associated with multiple myeloma. -Often affects multiple organ systems, including renal (nephrotic syndrome), cardiac (restrictive cardiomyopathy, arrhythmia), hematologic (easy bruising, splenomegaly), GI (hepatomegaly), and neurologic (neuropathy).

Back

Haemorrhagic Inflammation

Front

Back

Mast Cells

Front

Acute Inflammatory Cells

Back

pyknosis, karyorrhexis, karyolysis

Front

Back

oxalate crystals

Front

Back

Dystrophic calcification

Front

Back

Starvation Fatty Liver

Front

Back

caseous necrosis

Front

Back

Lipofuscin

Front

wear and tear pigment

Back

Eosinophils

Front

Acute Inflammatory Cells

Back

Diphtheritic Inflammation

Front

Back

Oak poisoning

Front

Tannins in Foal

Back

Glycogen Storage

Front

Back

Melanoma

Front

Back

Hydropic degeneration

Front

Back

Arteriosclerosis

Front

Hypertension

Back

Gangrene

Front

Back

Melanin

Front

Back

Fat Necrosis

Front

Back

Nutmeg Liver (Fatty Change)

Front

Back

Hyperpigmentation

Front

Back

Viral inclusion Bodies

Front

Back

Anthracosis

Front

Carbon

Back

Melanosis

Front

Back

Catarrhal Inflammation

Front

Back

Black disease (infectious necrotic hepatitis)

Front

Clostridium novae type B spores germinate in anaerobic environment created by liver flukes (Fasciola hepatica)

Back

Secondary Amyloidosis (AA)

Front

Back

Hepatic Amyloidosis

Front

Back

Basophils

Front

Acute Inflammatory Cells

Back

Liquefactive Necrosis

Front

Back

Endocrine Amyloidosis

Front

Back

Carotenoids

Front

Back

Atherosclerosis

Front

Rare in Animals

Back

Haemosiderin

Front

Can accumulate in vascular tissue as well

Back

Section 2

(45 cards)

Types of Post Mortem Changes

Front

Rigor Mortis, Algor Mortis, Clotting of Blood, Hypostatic Congestion (Livor Mortis), Post Mortem Imbibition of Blood, Post Mortem Imbibition of Bile, Gaseous Distentions, Autolysis (Self Digestion) Putrefaction

Back

Lymphocytosis

Front

increase in lymphocytes

Back

Plasma Cells

Front

Chronic Inflammatory Cells

Back

Crystal Formation

Front

Oxylate Crystals

Back

Fibroblasts and endothelial Cells

Front

Cells of tissue repair

Back

Benign vs. Malignant

Front

Benign - Uniform cellular appearance, normal mitotic figures, grow by expansion have clear boundaries Malignant - Invasive and Infiltrative, abnormal mitotic figures, major variation in cellular appearance, lack defined borders

Back

Neutropenia

Front

deficiency of neutrophils

Back

Causes of Malformation or Congenital Defects

Front

Chromosomal Defects, Viral Disease, Toxins, Drugs, Ionizing Radiation, Nutritional Deficiencies, Physical Factors, Female Anorexia

Back

Neutrophilia

Front

increase in neutrophils in blood

Back

Pneumonia

Front

Lung - Bronchopneumonia from air Interstitial pneumonia from hematogenous agents

Back

Lymphoplasmacytic Inflammation

Front

Inflammation of several layers around blood vessels (perivascular cuffing)

Back

Mastitis

Front

Inflammation of Mammary Gland

Back

Types of Necrosis

Front

Coagulative, Liquefactive, Caseous, Fat, Gangrenous

Back

Pancreatitis

Front

inflammation of the pancreas

Back

Purulent inflammation

Front

Back

Encephalitis

Front

Inflammation of the Brain

Back

Metastatic Cascade

Front

1. Primary Tumor (Detachment and Migration of Malignant cells) 2. Adhesion to vessel wall (Invade vessels) 3. Transport of micrometastasis 4. Evasion of host defenses and platelet clumping 5. Exit form vessel and migrate forming secondary tumor

Back

Macrophages

Front

Cells of Chronic Inflammation

Back

Granuloma

Front

central Core contains irritant and it is surrounded by chronic inflammatory cells

Back

Granulation Tissue

Front

Back

Prostatitis

Front

Inflammation of Prostate Gland

Back

Orchitis

Front

inflammation of the testes

Back

Mineralization

Front

Calcification

Back

Septicaemia

Front

Pathogenic Bacteria in Blood

Back

Non-infectious Arthritis

Front

Degenerative Joint Disease

Back

Cystitis

Front

Inflammation of Bladder

Back

Nephrotic Syndrome

Front

Glomerulonephritis or amyloidosis

Back

Eosinophilia

Front

increase in eosinophils

Back

Lymphocytes

Front

Cells of Chronic Inflammation

Back

Gastroenteritis

Front

Inflammation of GI Tract

Back

Monocytosis

Front

increase in monocytes

Back

Metritis

Front

Inflammation of Uterus

Back

Infectious Arthritis

Front

Back

Myelitis

Front

Inflammation fo spinal cord

Back

Nephritis

Front

Inflammation of Kidney

Back

Chronic Enteritis

Front

Back

Hepatitis

Front

Inflammation of Liver

Back

Types of Defects

Front

Neural Tube Defects, Skeletal Malformations, Polycystic Kidney, Skin Lesions, Muscular Defects, Cardiac Defects, Sexual Organ Malformation, Metabolic Disease (Rare)

Back

Bacteraemia

Front

Presence of non-pathogenic bacteria in blood

Back

Exogenous Pigementation

Front

Carbon (arthracosis), Dust (Pneumoconiosis), Carotenoids, Iatrogenic

Back

Types of Degenerations

Front

Hydropic, Lipidosis, Mucoid (Myxoid), Hyaline, Fibrinoid, Amyloid, Glycogen Storage, Cellular Inclusions

Back

Endogenous Pigmentation

Front

Melanin, Blood and Bile, Lipofuscin (Wear and Tear)

Back

Granulation Tissue Microscopically

Front

Back

Lymphopenia

Front

decrease in lymphocytes

Back

Cirrhosis

Front

Back